The relationship of exports, imports and economic growth between China and selected Asean countries

This study examines the relationship between the exports, imports and economic growth among China and the five selected ASEAN countries over the period of year 1993 to 2010. The annual time series data are collected from the Asian Development Bank and World Data Bank statistic databases in 2011. The variables used are includes the exports, imports, and GDP growth rate. The estimation methods of the study includes the Dickey Fuller Generalized Least Squares (DF-GLS) Unit Root Tests, Autoregressive Distributed Lag (ARDL) bound test, Error correction representation for ARDL model, and the Granger causality test. The empirical results show that the exports and imports between China and the five selected ASEAN countries contribute to the economic growth for those selected ASEAN countries which are Philippines, Vietnam, Indonesia, Thailand and Malaysia

Study of Bs→πρB_{s}\to \pi \rho decays in the perturbative QCD approach

In this note, we calculate the branching ratio and CP asymmetry parameters of $B_s\to\pi\rho$ in the framework of perturbative QCD approach based on $k_T$ factorization. This decay can occur only via annihilation diagrams in the Standard Model. We find that (a)the charge averaged $Br(B_s\to\pi^{+}\rho^{-}+\pi^{-}\rho^{+})$ is about $(9-12)\times10^{-7}$; $Br(B_s\to\pi^{0}\rho^{0}) \simeq 4\times10^{-7}$; and (b) there are sizable CP asymmetries in the processes, which can be tested in the near future Large Hadron Collider beauty experiments (LHC-b) at CERN or BTeV experiments at Fermilab.Comment: 6 pages, 3 figure

Spindle oscillations are generated in the dorsal thalamus and modulated by the thalamic reticular nucleus

Spindle waves occur during the early stage of slow wave sleep and are thought to arise in the thalamic reticular nucleus (TRN), causing inhibitory postsynaptic potential spindle-like oscillations in the dorsal thalamus that are propagated to the cortex. We have found that thalamocortical neurons exhibit membrane oscillations that have spindle frequencies, consist of excitatory postsynaptic potentials, and co-occur with electroencephalographic spindles. TRN lesioning prolonged oscillations in the medial geniculate body (MGB) and auditory cortex (AC). Injection of GABA~A~ antagonist into the MGB decreased oscillation frequency, while injection of GABA~B~ antagonist increased spindle oscillations in the MGB and cortex. Thus, spindles originate in the dorsal thalamus and TRN inhibitory inputs modulate this process, with fast inhibition facilitating the internal frequency and slow inhibition limiting spindle occurrence

血浆同型半胱胺酸和D-二聚体水平与急性期脑梗死各亚型的关系

Purpose-To study plasma homocysteine(Hcy)/D-dimer levels in patients with differernt subtype of acute cerebral infarction for early etiology diagnosis. Methods: The subjects were 246 acute cerebral infarction patients in the first 24 hours of stroke onset at our hospital. We divided them into large –artery atherosclerosis(LAA) group(n=66), small artery occlusion(SAO) group(n=76), cardio embolism(CE) group(n=40),stroke of other demonstrated etiology(SOE) group (n=11)and stroke of other undemonstrated etiology(SUE) group(n=51) according to the TOAST classification. Plasma homocysteine and D-diamer were determined within the first 24 hours of admission, the differences in subgroups and control group(n=121) were analyzed. Results:Plasma homocysteine levels in LAA group were signficantly higher than other groups(P<0.05), no differences in non-LAA groups(P>0.05);Plasma D-diamer levels in CE group were significant higher than other groups(P<0.05), no differences in non-CE groups(P>0.05). Conclusions: Plasma homocysteine and D-diamer level are associated with subtypes of acute cerebral infarction, and it contributed to early classification diagnosis and individual treatment of patients with acute cerebral infarction.目的：探讨血浆同型半胱胺酸和D-二聚体在急性期各亚型脑梗死之间的差异，为临床脑梗死亚型的病因学诊断提供理论依据。方法：收集我院246例24 h内发病的脑梗死患者，根据TOAST分型方法进行病因学分型：大动脉粥样硬化性脑卒中(1arge—artery atherosclerosis，LAA；n=66)、小动脉闭塞性脑卒中(small-artery occlusion ,SAO；n=76)、心源性脑栓塞(cardio embolism，CE；n=40)、其他确定原因引发的缺血性脑卒中(stroke of other demonstrated etiology，SOE；n=11)和不明原因的缺血性脑卒中(stroke of other undemonstrated etiology，SUE；n=51)。在入院24小时内测定血浆同型半胱胺酸（Hcy）和D-二聚体（DD）的含量，并与121例对照组进行比较，分析二者在不同亚型脑梗死及对照组之间的差异。结果：LAA组同型半胱氨酸水平明显高于其它4组及对照组(P<0．05) ，其他 4个亚型(SAA、CA、SOE、SUE)及对照组组间比较差异无统计学意义(P>0．05)。CE组血浆D-二聚体含量最高,显著高于其他各亚型组及对照组(P<0.05); 其他 4个亚型(SAA、CA、SOE、SUE)及对照组组间比较差异无统计学意义(P>0．05)。结论：急性期各亚型脑梗死之间血浆同型半胱胺酸和D-二聚体升高程度不同,这对于临床早期分型诊断及个体化治疗有着重要指导意义

Detection and characterization of spontaneous internal deletion mutants of Beet Necrotic yellow vein virus RNA3 from systemic host Nicotiana benthamiana

Abstract Background Beet Necrotic Yellow Vein virus (BNYVV) is a member of the genus Benyvirus causing a worldwide sugar beet disease rhizomania. BNYVV contains four or five plus-sense single stranded RNAs. In altered selective conditions, multipartite RNA viruses of plant are prone to undergoing internal deletions, thus turning into Defective RNAs (D RNAs). Although several D RNAs have been reported in BNYVV infection, the spontaneous internal deletion mutants responsible for severe symptom in systemic host Nicotiana benthamiana (N. benthamiana) are not described so far. Results Systemic host N. benthamiana was inoculated by Chinese BNYVV isolates. RT-PCR and Northern blot showed that the D RNAs forms of BNYVV RNA3 were present in the systemic infection of the N. benthamiana. Three distinct D-RNA3s, named as D-RNA 3α, D-RNA 3β and D-RNA 3γ, were made into infectious clones. When inoculated on the N. benthamiana, the in vitro transcripts of D forms exhibited more stable than that of wild-type RNA3 in systemic movement. Among the detected mutant, the p25 protein frame-shift mutant (D-RNA3α) induced obvious necrotic lesions on Tetragonia.expansa (T. expansa) and pronounced systemic symptom on the N. benthamiana. The D-RNA3α was further mutated artificially to pre-terminate the downstream N protein, leading to the abolishment of the pathogenicity, indicating the N protein was responsible for the necrotic symptom. Conclusion Our studies demonstrated the internal deletion mutants of BNYVV-RNA3 were spontaneously generated in the systemic infection on N. benthamiana. The internal deletions didn't affect the efficient replication of D-RNA3s, instead by improving the stability and pathogenicity of RNA3 in the systemic host N. benthamiana. Besides, our results also suggested the downstream N protein of RNA3, but not the upstream p25 protein, may play an important role in the systemic infection on N. benthamiana

Bis[4-(2-hy­droxy­benzyl­idene­amino)­benzoato-κO 1]tetra­kis­(methanol-κO)cadmium

In the title mononuclear complex, [Cd(C14H10NO3)2(CH3OH)4], the Cd2+ cation is situated on an inversion centre. It exhibits a distorted octa­hedral coordination, defined by two carboxyl­ate O atoms from two monodentate anions and by four O atoms from four methanol mol­ecules. The crystal structure comprises intra­molecular O—H⋯O and O—H⋯N, and inter­molecular O—H⋯O hydrogen bonds. The latter help to construct a layered structure extending parallel to (100)